Adenosine deaminase (ADA) deficiency accounts for about half of the autosomal recessive forms of SCIDs. ADA follows PNP in purine nucleoside catabolism, but deficiency in this enzyme causes even more severe symptoms than PNP deficiency, which is a T cell deficiency. ADA degrades toxic adenosine and deoxyadenosine, which accumulate in the cells of patients. Immature lymphoid cells are particularly sensitive to these nucleotides. In addition to immunological defect, most patients with ADA deficiency also have skeletal abnormalities.
Alternative names
Adenosine aminohydrolase, Severe combined immunodeficiency due to adenosine deaminase deficiency
SCID Due to ADA deficiency
ADA-SCID
Classification
- Combined B and T cell immunodeficiencies
- Deficiencies of purine metabolism
Inheritance
Autosomal recessive
Incidence
1/2 x 100 to 1 x 100 births.