Defects in RAC2 are the cause of neutrophil immunodeficiency syndrome, that clinically resembles LAD1. Rac2 is part of the NADPH oxidase complex. Rac2 is necessary for the regulation of the actin cytoskeleton (chemotaxis and degranulation) and NADPH oxidase (superoxide production) function.
Alternative names
LAD caused by Rac2 deficiency
Neutrophil immunodeficiency syndrome
Classification
- Defects of phagocyte function
- Leukocyte adhesion defects
Inheritance
Autosomal dominant
Cross references
Phenotypically related immunodeficiencies
Incidence
Incidence is not known yet.