Properdin is synthesized by monocytes, hepatocytes, and T cells, and it is a component of the secondary granule in neutrophiles. In some families, properdin is undetectable, while in other affected males may have levels as high as 10% of normal. In other kindreds the serum level of properdin are normal but the protein is dysfunctional. Three phenotypes have been reported: complete deficiency (type I), incomplete deficiency (type II), and dysfunction of properdin (type III).
Alternative names
Properdin P factor deficiency
Classification
- Defects of the alternative complement pathway
Inheritance
X-linked
Cross references
Phenotypically related immunodeficiencies
Incidence
Incidence is not known.