Defects in RAB27A cause Griscelli syndrome type 2 (GS2). RAB27A regulates the cytotoxic granule exocytosis and affect T-lymphocyte and macrophage-activation. RABD27 encodes protein which is key effector of intracellular vesicular transport. Most patients also develop an uncontrolled T lymphocyte and macrophage activation syndrome, known as hemophagocytic syndrome, leading to death in the absence of bone marrow transplantation.
Alternative names
GS2
Griscelli syndrome with hemophagocytic syndrome
PAID
Partial albinism with immunodeficiency
Classification
- Defects of phagocyte function
- Griscelli syndrome
Inheritance
Autosomal recesseive
Cross references
Phenotypically related immunodeficiencies
IDR factfile for Chediak-Higashi syndrome
Incidence
Incidence is not known.